File:Regulation of reactive oxygen species and e-cigarette-induced oxidative damage and mitochondrial dysfunction.png
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Size of this preview: 800 × 501 pixels. Other resolutions: 320 × 201 pixels | 640 × 401 pixels | 1,024 × 642 pixels | 1,280 × 802 pixels | 3,203 × 2,007 pixels.
Original file (3,203 × 2,007 pixels, file size: 1.01 MB, MIME type: image/png)
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 10:56, 28 March 2024 | | 3,203 × 2,007 (1.01 MB) | commons>Ameisenigel | {{Information |Description=The regulation of reactive oxygen species. Under normal conditions, glutathione functions in scavenging reactive oxygen species, and levels of NRF2 remain low due to ubiquitin-mediated degradation in the proteasome (left panel). If oxidative stress increases, NRF2 escapes protein degradation upon phosphorylation and separation from KEAP1, and it enters the nucleus to regulate gene expression by binding the antioxidant response element (ARE) as a normal reaction in r... |
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