|Other names: Hypermetropia, hyperopia, longsightedness, long-sightedness|
|Far-sightedness without (top) and with lens correction (bottom)|
|Symptoms||Close objects appear blurry|
|Complications||Accommodative dysfunction, binocular dysfunction, amblyopia, strabismus|
|Causes||Too short an eyeball, misshapen lens or cornea|
|Risk factors||Family history|
|Diagnostic method||Eye exam|
|Differential diagnosis||Amblyopia, retrobulbar optic neuropathy, retinitis pigmentosa sine pigmento|
|Treatment||Eyeglasses, contact lenses, surgery|
Far-sightedness, also known as hypermetropia, is a condition of the eye in which light is focused behind, instead of on, the retina. This results in close objects appearing blurry, while far objects may appear normal. As the condition worsens, objects at all distances may be blurry. Other symptoms may include headaches and eye strain. People may also experience accommodative dysfunction, binocular dysfunction, amblyopia, and strabismus.
The cause is an imperfection of the eyes. Often it occurs when the eyeball is too short, or the lens or cornea is misshapen. Risk factors include a family history of the condition, diabetes, certain medications, and tumors around the eye. It is a type of refractive error. Diagnosis is based on an eye exam.
Management can occur with eyeglasses, contact lenses, or surgery. Glasses are easiest while contact lenses can provide a wider field of vision. Surgery works by changing the shape of the cornea. Far-sightedness primarily affects young children, with rates of 8% at 6 years and 1% at 15 years. It then becomes more common again after the age of 40, affecting about half of people.
Signs and symptoms
The signs and symptoms of far-sightedness are blurry vision, headaches, and eye strain. The common symptom is eye strain. Difficulty seeing with both eyes (binocular vision) may occur, as well as difficulty with depth perception.
Simple hypermetropia, the commonest form of hypermetropia is caused by normal biological variations in the development of eyeball. Aetiologically, causes of hypermetropia can be classified as:
- Axial: Axial hypermetropia occur when the axial length of eyeball is too short. About 1 mm decrease in axial length cause 3 diopters of hypermetropia. One condition that cause axial hypermetropia is nanophthalmos.
- Curvatural: Curvatural hypermetropia occur when curvature of lens or cornea is flatter than normal. About 1 mm increase in radius of curvature results in 6 diopters of hypermetropia. Cornea is flatter in microcornea and cornea plana.
- Index: Age related changes in refractive index (cortical sclerosis) can cause hypermetropia. Another cause of index hypermetropia is diabetis. Occasionally, mild hypermetropic shift may be seen in association with cortical or subcapsular cataract also.
- Positional: Positional hypermetropia occur due to posterior dislocation of Lens or IOL.
- Consecutive: Consecutive hypermetropia occur due to surgical over correction of myopia or surgical under correction in cataract surgery.
- Functional: Functional hypermetropia results from paralysis of accommodation as seen in internal ophthalmoplegia, CN III palsy etc.
- Absence of lens: Congenital or acquired aphakia cause high degree hypermetropia.
Far-sightedness is often present from birth, but children have a very flexible eye lens, which helps to compensate. In rare instances hyperopia can be due to diabetes, and problems with the blood vessels in the retina.
A diagnosis of far-sightedness is made by utilizing either a retinoscope or an automated refractor-objective refraction; or trial lenses in a trial frame or a phoropter to obtain a subjective examination. Ancillary tests for abnormal structures and physiology can be made via a slit lamp test, which examines the cornea, conjunctiva, anterior chamber, and iris.
In severe cases of hyperopia from birth, in regards to preterm infants seem to have a higher incidence. A child with severe hyperopia can't see objects in detail. If the brain never learns to see objects in detail, then there is a high chance of one eye becoming dominant. The result is that the brain will block the impulses of the non-dominant eye. 
Hyperopia is typically classified according to several aspects, there are three clinical categories of hyperopia.
- Simple hyperopia: Occurs naturally due to biological diversity.
- Pathological hyperopia: Caused by disease, trauma, or abnormal development.
- Functional hyperopia: Caused by paralysis that interferes eye's ability to accommodate.
Classification according to severity
There are also three categories severity:
- Low: Refractive error less than or equal to +2.00 diopters (D).
- Moderate: Refractive error greater than +2.00 D up to +5.00 D.
- High: Refractive error greater than +5.00 D.
- Total hypermetropia: It is the total amount of hyperopia which is obtained after complete relaxation of accommodation using cycloplegics like atropine.
- Latent hyperopia: It is the amount of hyperopia normally corrected by ciliary tone (approximately 1 diopter).
- Manifest hyperopia: It is the amount of hyperopia not corrected by ciliary tone. Manifest hyperopia is further classified into two, facultative and absolute.
- Facultative hyperopia: It is the part of hyperopia corrected by patient's accommodation.
- Absolute hyperopia: It is the residual part of hyperopia which causes blurring of vision for distance.
So, Total hyperopia= latent hyperopia + manifest hyperopia (facultative + absolute)
- Photorefractive keratectomy (PRK): This is a refractive technique that is done by removal of a minimal amount of the corneal surface. Hyperopic PRK has many complications like regression effect, astigmatism due to epithelial healing, and corneal haze. Post operative epithelial healing time is also more for PRK.
- Laser assisted in situ keratomileusis (LASIK): Laser eye surgery to reshape the cornea, so that glasses or contact lenses are no longer needed. Excimer laser LASIK can correct hypermetropia upto +6 diopters. LASIK is contraindicated in patients with lupus and rheumatoid arthritis.
- Laser epithelial keratomileusis (LASEK): Resembles PRK, but uses alcohol to loosen the corneal surface.
- Epi-LASIK: Epi-LASIK is also used to correct hyperopia. In this procedure, use of epikeratome eliminates the use of alcohol.
- Laser thermal keratoplasty (LTK): Laser thermal keratoplasty is a laser based non-destructive refractive procedure used to correct hyperopia and presbyopia. It uses Thallium-Holmium-Chromium (THC): YAG laser.
- Ahakia correction: High degree hypermetropia due to absence of lens (aphakia) is best corrected using intraocular lens implantation.
- Refractive lens exchange (RLE): A variation of cataract surgery where the natural crystalline lens is replaced with an artificial intraocular lens; the difference is the existence of abnormal ocular anatomy which causes a high refractive error.
- Phakic IOL: Phakic intraocular lens are lenses that implanted inside eye without removing the normal crystalline lens. Phakic IOLs can be used to correct hypermetropia upto +20 diopters.
Non laser procedures
- Conductive keratoplasty (CK): Conductive keratoplasty is a non laser refractive procedure used to correct presbyopia and low hypermetropia (+0.75D to +3.25D) with or without astigmatism (upto 0.75D). It uses radiofrequency energy to heat and shrink corneal collagen tissue. CK is contraindicated in pregnant/breastfeeding women, central corneal dystrophies and scarring, history of herpetic keratitis, type 1 diabetes etc.
- Automated lamellar keratoplasty (ALK): Hyperopic automated lamellar keratoplasty (H-ALK) and Homoplastic ALK are ALK procedures that corrects low to moderate hyperopia. Poor predictability and the risk of complications limits usefulness of these procedures.
- Keratophakia and epi-keratophakia are another two non laser surgical procedures used to correct hypermetropia. Keratophakia is a surgical technique developed by Barraquer for treating high hypermetropia and aphakia. Poor predictability and induced irregular astigmatism are complications of these procedures.
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